'Lowprotein\r'

'Not all course occurring metals are used by the torso for important biological processes. Lead and gougedle are examples of these metals that are not infixed for life but may purge cause perniciousness and death if taken in large doses. A occupy conducted in Japan showed a correlation between protein intake with the increased vulnerability for intoxication of compact disk (Tavari 1986). Rats given a humble protein diet were observed to present high toxicity from these metals that were also sight in the urine and feces (Suzuki 1984).  A low protein diet in adult male is usually advised to those with kidney and liver diseases and as a result, this negative effect of higher metal toxicity is important to understand.\r\nMetals mask by covert to organic compounds afterwards altering their structure and perchance modifying their scat. When the function in not carried extinct well, this discharge lead to cell death and defusing of the production of important enzym es (â€Å"Metals as toxins”). For instance, a metal compound can debate with a biologically momentous fraction such as oxygen to gain an enzyme responsible for degrading glucose. If this metal successfully defeats oxygen, the enzyme may not be produced; thus, glucose exit not be degraded and possibly accumulated. This is a simple example of what a metal can do to the body.\r\nIn the case of normal protein intake, the body has lavish proteins that can bind to harmful compounds such as metals. Similar to the action of a lock and key, a specific protein can complex with damaging bioelements and then clear them outside the body to prevent attainable internal damage. Metallothionein specifically works as a chelating agent and combines itself with cadmium, for example and is excreted out of the body while Selenium, a protein profusely found in egg whites, suppresses the toxic effect of metals (â€Å"Metals as toxins”). A low protein intake thus, have a significant effect in resulting to high levels of cadmium and lead in the blood since nigh protein clearly function in brush out these harmful metals by binding with them and then carrying them out of the body.\r\nReferences\r\nâ€Å"Metals as toxins.” Retrieved rarefied 11, 2007, from\r\nhttp://www.portfolio.mvm.ed.ac.uk/studentwebs/session2/group29/introtox.htm\r\nSuzuki, K.T., Miyamoto, E., Tanaka, Y. Kawamura, R. and Yamamura, M. (1984). Effect of diet\r\non urinary and fecal excretion of cadmium, copper and coat from rats preaccumulated\r\nheavily with cadmium. Archives of Environmental befoulment and Toxicology, vol.\r\n13 no.5. Retrieved August 11, 2007, from\r\nwww.springerlink.com/index/J456157JVU87212T.pdf\r\nTavari, P.C., Jain, V.K., Ashquin, M. and Tandon, S.K. (1986). bias of protein deficiency\r\non cadmium toxicity in rats. Archives of Environmental contaminant and Toxicology,\r\nvol.15 no.4. Retrieved August 11, 2007, from\r\nwww.springerlink.com/index/G88L4P84417X T467.pdf\r\n \r\n \r\n'